Following a primary infection, the virus enters the nerves at the site of primary infection, migrates to the cell body of the neuron, and becomes latent in the ganglion.As a result of primary infection, the body produces antibodies to the particular type of HSV involved, preventing a subsequent infection of that type at a different site.More serious disorders occur when the virus infects and damages the eye (herpes keratitis), or invades the central nervous system, damaging the brain (herpes encephalitis).

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HSV infection has also been associated with cognitive deficits of bipolar disorder, and Alzheimer's disease, although this is often dependent on the genetics of the infected person.

In all cases, HSV is never removed from the body by the immune system.

Herpetic sycosis is a recurrent or initial herpes simplex infection affecting primarily the hair follicle.

This theory has been contested, however, since HSV is detected in large numbers of individuals having never experienced facial paralysis, and higher levels of antibodies for HSV are not found in HSV-infected individuals with Bell's palsy compared to those without.

Hi, I was diagnosed with genital herpes type 2 a couple of weeks ago.

My first outbreak was horrific but the sores cleared up within 5 days on aciclovir.HSV asymptomatic shedding occurs at some time in most individuals infected with herpes.It can occur more than a week before or after a symptomatic recurrence in 50% of cases.Many people infected with HSV-2 display no physical symptoms—individuals with no symptoms are described as asymptomatic or as having subclinical herpes.Herpes whitlow is a painful infection that typically affects the fingers or thumbs.When infecting the brain, the virus shows a preference for the temporal lobe.