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The most famous use is surely the space bridge, which allows a traveller to traverse vast intergalactic distances almost instantaneously by means of a "short-cut" through transwarp space.As transwarp space exists beyond the laws of reality, it is even possible to travel through time itself by crossing it.Other research focused on the enhancement of photocatalysis by modification of by means of metal loading, metal ion doping, dye sensitization, composite semiconductor, anion doping and metal ion-implantation.
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Though not often discussed in detail, this method of transit was in play from virtually the very beginning of Transformers fiction: space bridges explicitly operated on the principle in both the original The Transformers cartoon and Marvel comic, with the dangers of the interdimensional void occasionally serving as plot points.
Many subsequent Generation 1 stories would feature pieces of technology that operated on this system, with many different names for the interdimensional space involved being used.
Eventually, subspace was canonized, by name, in 2004 by the Story of Binaltech, which established it as the void between dimensions, and saw characters travel through it to reach separate dimensions.
In 1996, the Beast Wars cartoon introduced the world to transwarp technology.
The ultimate expression of the technology came with the development of the portable Transwarp Drive, powered by transwarp cells, which removed the need for a fixed departure/arrival location system and allowed whole vessels to independently voyage across time and space at will.
However, its widespread use does not necessarily mean that transwarp space has a glowing reputation.transcatheter particulate embolization of bronchial arteries using polyvinyl alcohol (PVA) Gelfoam pledgets (effective in 70 95%, but recurrent bleeding in 20 30% of patients) N.B.: search for artery of Adamkiewicz at vertebral level of T8 to L2 prior to embolization(a) forced inspiration causes a high negative intrathoracic pressure (M ller maneuver) and increases venous return (b) obstruction creates high positive intrathoracic pressure that impairs development of edemastereotypical inflammatory response of alveolar wall to injury (a) acute phase: fluid inflammatory cells exude into alveolar space, mononuclear cells accumulate in edematous alveolar wall (b) organizing phase: hyperplasia of type II pneumocytes attempt to regenerate alveolar epithelium, fibroblasts deposit collagen (c) chronic stage: dense collagenous fibrous tissue remodels normal pulmonary architecture: hamartoma (6%, 3rd most common lung mass), chondroma : lipoma (usually pleural lesion) : fibroma : leiomyoma : schwannoma, neurofibroma, paraganglioma : intrapulmonary lymph node : amyloid, splenosis, endometrioma, extramedullary hematopoiesisincreases lung compliance, stabilizes alveoli, enhances alveolar fluid clearance, reverses surface tension, protects against alveolar collapse during respiration, protects epithelial cell surface, reduces opening pressure precapillary tone HIV retrovirus attaches to CD4 molecule on surface of T-helper lymphocytes macrophages microglial cells; after cellular invasion HIV genetic information is incorporated into cell's chromosomal DNA; virus remains dormant for weeks to years; after an unknown stimulus for viral replication CD4 lymphocytes are destroyed (normal range of 800 1,000 cells/mmendobronchial fungal proliferation followed by transbronchial vascular invasion eventually causes widespread hemorrhage thrombosis of pulmonary arterioles ischemic tissue necrosis systemic dissemination; fungus ball = devitalized sequestrum of lung infiltrated by fungiacid rapidly disseminates throughout bronchial tree lung parenchyma, incites a chemical pneumonitis within minutes; extent of injury from mild bronchiolitis to hemorrhagic pulmonary edema depends on p H aspirated volumesubmucosal and peribronchiolar fibrosis = irreversible fibrosis of small airway walls with narrowing/obliteration of airway lumina (respiratory bronchiole, alveolar duct, alveoli) by granulation tissue of immature fibroblastic plugs (Masson bodies)(1) Bacterial/fungal pneumonia (response to antibiotics, positive cultures) (2) Chronic eosinophilic pneumonia (young female, eosinophilia in 2/3) (3) Usual interstitial pneumonia (irregular opacities, decreased lung volume)postobstructive pneumonia, organizing adult respiratory distress syndrome, lung cancer, extrinsic allergic alveolitis, pulmonary manifestation of collagen vascular disease, pulmonary drug toxicity, silo filler disease, idiopathic (50%)granulation tissue polyps filling the lumina of alveolar ducts and respiratory bronchioles (bronchiolitis obliterans) variable degree of infiltration of interstitium and alveoli with macrophages (organizing pneumonia)subtype of well-differentiated adenocarcinoma; cuboidal/columnar cells grow along alveolar walls septa without disrupting the lung architecture or pulmonary interstitium (serving as scaffolding for tumor growth)necrotizing granulomas surrounding small airways; pulmonary arteritis as a secondary phenomenon (1) large masses of eosinophils in necrotic zones, associated with endobronchial mucus plugs, eosinophilic pneumonia, Charcot-Leyden crystals, fungal hyphae in granulomas (with asthma) (2) polymorphonuclear cell infiltrate in necrotic zones (without asthma)small uniform oval cells with scant cytoplasm; nuclei with stippled chromatin; numerous mitoses large areas of necrosis; in 20% coexistent with non-small cell histologic types (most frequently squamous cell)bronchiectasis, lung abscess, empyema, bronchial atresia, congenital lobar emphysema, cystic adenomatoid malformation, intrapulmonary bronchogenic cyst, Swyer-James syndrome, pneumonia, arteriovenous fistula, primary/metastatic neoplasm, hernia of Bochdalekresembles normal lung with diffuse dilatation of bronchioles alveolar ducts alveoli; dilatation of subpleural peribronchiolar lymph vessels; covered by mesothelial layer overlying fibrous connective tissue; congenital cystic adenomatoid malformation type ii is present in 15 25%intra- and perivascular granulomas, intimal hyperplasia, medial hypertrophy, concentric collagen deposition and fibrosis of vessel walls; localized alveolitis with eosinophilic infiltration; pulmonary infarction(a) acute phase: intraalveolar, intrabronchial, peribronchial, interstitial accumulation of inflammatory cells edema(b) chronic phase (1 2 weeks after initial onset): proliferative bronchiolitis, parenchymal fibrosis, pneumatocele formation(1) Compression of SVC (64%) pulmonary veins (4%) (2) Chronic obstructive pneumonia (narrowing of trachea/central bronchi) in 5% (3) Esophageal stenosis (3%) (4) Pulmonary infarcts fibrosis (narrowing of pulmonary artery) (5) Prominent intercostal arteries (narrowing of pulmonary artery)enters thorax through aortic hiatus; ascends in right prevertebral location (between azygos vein descending aorta); swings to left at T4 6 posterior to esophagus; ascends for a short distance along right of aorta; crosses behind aortic arch; runs ventrally at T3 between left common carotid artery left subclavian artery(1) Thoracentesis (leading to loss of calories, lymphocytopenia, hypogammaglobulinemia) (2) Total parenteral nutrition (3) Thoracic duct ligation (if drainage exceeds 1,500 m L/day for adults or 100 m L/yr-age/day for children proliferation of bronchial structures at the expense of alveolar saccular development, modified by intercommunicating cysts of various size (adenomatoid overgrowth of terminal bronchioles, proliferation of smooth muscle in cyst wall, absence of cartilage)(1) Congenital lobar emphysema (2) Diaphragmatic hernia (3) Bronchogenic cyst (small solitary cyst near midline) (4) Neurenteric cyst (5) Bronchial atresia (6) Bronchopulmonary sequestration (less frequently associated with polyhydramnios/hydrops) (7) Mediastinal/pericardial teratoma1:2,000 1:2,500 livebirths; almost exclusively in Caucasians (5% carry a CF mutant gene allele); unusual in Blacks (,000), Orientals, Polynesians The most common inherited disease among Caucasian Americans!" When the Autobot leader transformed into robot mode in the original cartoon, his trailer would almost always slide out of the frame, not to be seen again until he transformed back into truck mode, at which point it would come sliding in to connect back up with him.Fans warmed to the notion that it was being shunted into this subspace, where it remained until Prime summoned it back, a concept that also helped to explain the assorted cases of size changing evident in Transformers fiction, with the notion that characters who shrank when they transformed did so by shunting their extraneous mass into subspace.Sandwiched between the infinite universal streams that make up the multiverse, there is an expanse of nothingness that acts as a buffer between dimensions.